fig3

Beta2-glycoprotein I cooperate with hepatitis B surface antigen promotes hepatocellular carcinogenesis via the nuclear factor kappa B signal pathway were enhanced by the lipopolysaccharide

Figure 3. Expression of TNF-α, IL-1β and AFP by ELISA in six groups. Group A: non-transfected, non-treated cells; group B: transient β2GPI-and HBsAg-transfection without LPS treatment; group C: non-transfected cells treated with 100 ng/mL LPS; group D: transient β2GPI- and HBsAg-transfection and treated with 100 ng/mL LPS; group E: transient β2GPI-transfection and treated with 100 ng/mL LPS; group F: transient HBsAg-transfection and treated with 100 ng/mL LPS. Data presented as means ± SD. a: groups B, C, D, E, and F compared with group A, P < 0.05; b: group B compared with groups A, C, E, and F, P < 0.05; c: groups E and F compared with group C, P < 0.05; d: group D compared with groups A, B, C, E, and F, P < 0.00; e: group E compared with group F, P > 0.05; f: group C compared with groups B, E, and F, P < 0.05. β2GPI: beta2-glycoprotein I; HBsAg: hepatitis B surface antigen; LPS: lipopolysaccharide

Hepatoma Research
ISSN 2454-2520 (Online) 2394-5079 (Print)

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